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Icity of the liposomal surface, and/or by encapsulating additional adjuvantsIcity of the liposomal surface, and/or by encapsulating additional adjuvants ("conventional" and second-generation liposomes, the [44-46] stealth technology ). Since liposomes were first described in 1960, these nanoparticulate carriers were investigated for various purposes - including industrial, pharmaceutical, cl
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Ained parallel sections with a pooled IgG control. This protein-level confirmation of our microarray data gave us the impetus to pursue functional in vitro and in vivo assays with galectin-1 over-expressing GBM cells.Extracellular matrix attachmentResultsIdentification of galectin-1 as a potential mediator of glioma invasionThe quantity of RNA obtained from various xenograft tumors was highly vari
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T and persistent Ab responses to beta-amyloid; the Ab response wasT and persistent Ab responses to beta-amyloid; the Ab response was characterized by a marked prevalence of IgG1 over the IgG2a isotype Ref. [65]BS1-EMimotope 1 from HIV-1 bridging sheet domain (BS) HIV-1 SF162 Env V3 loop peptide 291-336 from gp120 (HXB2 numbering) HIV-1 SF162 Env MPER peptide 649-689 from gp41 (HXB2 numbering
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Combine to form a C5 convertase. Runaway complement activation is prevented by binding of Complement Receptor 1 (CR1) and a constitutively active membrane bound Decay Accelerating Factor (DAF, or CD55) which can prevent the complement cascade[51]. In patients with severe dengue, large amounts of C3a have been detected revealing a role for complement in dengue pathogenesis. This finding might be an
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